PRESSURE ULCER (BED SORE) TREATMENT

Definition

Tissue damage and ulceration caused by prolonged pressure. Also referred to as bed sores, decubitus ulcers, pressure sores, trophic ulcers, or penetrating ulcers.

Preventable

Pressure ulcers are preventable with proper care and management.

Incidence

  • Affects about 5% of hospitalized patients.
  • Higher incidence in paraplegic patients, the elderly, and those with severe illnesses.

Most Common Sites

Ischium > Greater trochanter > Sacrum > Heel > Malleolus > Occiput.

Mechanism

External pressure exceeding capillary occlusive pressure (over 30 mm Hg) leads to interrupted blood flow, causing tissue hypoxia, necrosis, and ulceration.

Neurological Causes

Conditions such as:

  • Syringomyelia
  • Spina bifida
  • Spinal injuries
  • Peripheral neuritis
  • Peripheral nerve injury
  • Leprosy
  • Diabetic neuropathy
  • Paraplegia
  • Tabes dorsalis

Clinical Features

  • Painless, well-defined ulcers.
  • Base of the ulcer is often formed by bone.

STAGING OF PRESSURE SORES

StageDescription
INon-blanchable redness without skin break (early superficial ulcer).
IIPartial skin loss involving epidermis and dermis (late superficial ulcer).
IIIFull skin loss extending into subcutaneous tissue but not through fascia (early deep ulcer).
IVFull skin loss through fascia with extensive damage, potentially involving muscle, bone, tendon, or joint (late deep ulcer).

Management

General Principles

  • Maintain ABCD&E (Airway, Breathing, Circulation, Disability, Exposure).
  • Prevention is the best treatment.

Preventive Measures

  • Good skin care.
  • Use of special pressure dispersion cushions or foams.
  • Use of low air-loss and air-fluidized beds.
  • Urinary or fecal diversion in selected cases.

For Bed-Bound Patients

  • Reposition the patient at least every 2 hours.

For Wheelchair-Bound Patients

  • Lift themselves off their seat for 1 second every 10 minutes.

Surgical Treatment

  • Reserved for patients with no improvement after conservative management.
  • Includes:
    • Adequate debridement.
    • Vacuum-assisted closure.
    • Flap closure.

VACUUM-ASSISTED CLOSURE/NEGATIVE PRESSURE WOUND THERAPY (NPWT)

How NPWT Works

  • Promotes wound healing by applying a vacuum through a special sealed dressing.
  • The vacuum removes fluid from the wound and increases blood flow to the area.
  • Vacuum can be applied continuously or intermittently, depending on the wound type and clinical objectives.
  • A negative pressure of -125 mm Hg is typically used.

Dressing Changes

  • Dressing should be changed 2-3 times per week.

Primary Effects of NPWT on Wound Healing

EffectDescription
MacrodeformationDraws wound edges together, promoting closure.
MicrodeformationStimulates cellular proliferation on the wound surface.
Stabilization of Wound EnvironmentProtects the wound from external microbes in a warm, moist environment.
Reduced EdemaRemoves excess fluid from soft tissues.

Contraindications for NPWT Use

  • Malignancy in the wound.
  • Untreated osteomyelitis.
  • Non-enteral and unexplored fistulas.
  • Necrotic tissue with eschar.

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